Parkinson's disease (PD) is a common neurodegenerative disorder with motor and non-motor manifestations such as impaired balance, gait disturbances and progressive loss of mobility. Bone involvement is common in patients with PD, including low bone mass and an increased risk of fractures, especially hip fractures. Increased risk of falls results from impaired balance, gait disorders, cognitive dysfunction, and autonomic failure. Osteoporosis in PD results from nutritional deficiencies, vitamin D deficiency, weight loss with sarcopenia, and progressive muscle weakness. Antiparkinsonian drugs, such as levodopa, contribute to osteoporosis through hyperhomocysteinemia. In addition, dopamine depletion and chronic inflammation disrupt bone remodeling. The review summarizes the evidence on bone mineral density, bone quality, falls and fractures in PD and discusses the pathophysiological mechanisms of this comorbidity.